CSHL study shows that some malignant tumors can be shut down after all

(Cold Spring Harbor Laboratory) More than half of all human cancers have mutations that disable a protein called p53. When cells lose p53, tumors grow aggressively and often cannot be treated. But a new study by Alea Mills and colleagues from CSHL may offer a way to counteract the problem. The scientists have succeeded in shutting off the growth of p53-deficient tumors by turning up the production of TAp63 proteins, a class of proteins produced by the p63 gene.

source : www.eurekalert.org

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Submited at Monday, November 9th, 2009 at 1:00 pm on Cancer and Oncology by ethan
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